Full metadata record
DC Field | Value | Language |
---|---|---|
dc.contributor.author | 강덕희 | * |
dc.contributor.author | 최규복 | * |
dc.contributor.author | 김승정 | * |
dc.contributor.author | 류동열 | * |
dc.date.accessioned | 2016-08-28T12:08:16Z | - |
dc.date.available | 2016-08-28T12:08:16Z | - |
dc.date.issued | 2011 | * |
dc.identifier.issn | 1011-8934 | * |
dc.identifier.other | OAK-7997 | * |
dc.identifier.uri | https://dspace.ewha.ac.kr/handle/2015.oak/221969 | - |
dc.description.abstract | We have hypothesized that non-dipper status and left ventricular hypertrophy (LVH) are associated with the development of chronic kidney disease (CKD) in non-diabetic hypertensive patients. This study included 102 patients with an estimated glomerular filtration rate (eGFR) ≥ 60 mL/min/1.73 m2. Ambulatory blood pressure monitoring and echocardiography were performed at the beginning of the study, and the serum creatinine levels were followed. During the average follow-up period of 51 months, CKD developed in 11 patients. There was a significant difference in the incidence of CKD between dippers and non-dippers (5.0% vs 19.0%, P < 0.05). Compared to patients without CKD, patients with incident CKD had a higher urine albumin/creatinine ratio (52.3 ± 58.6 mg/g vs 17.8 ± 29.3 mg/g, P < 0.01), non-dipper status (72.7% vs 37.4%, P < 0.05), the presence of LVH (27.3% vs 5.5%, P < 0.05), and a lower serum HDL-cholesterol level (41.7 ± 8.3 mg/dL vs 50.4 ± 12.4 mg/dL, P < 0.05). Based on multivariate Cox regression analysis, non-dipper status and the presence of LVH were independent predictors of incident CKD. These findings suggest that non-dipper status and LVH may be the therapeutic targets for preventing the development of CKD in non-diabetic hypertensive patients. © 2011 The Korean Academy of Medical Sciences. | * |
dc.language | English | * |
dc.title | Non-dipper status and left ventricular hypertrophy as predictors of incident chronic kidney disease | * |
dc.type | Article | * |
dc.relation.issue | 9 | * |
dc.relation.volume | 26 | * |
dc.relation.index | SCI | * |
dc.relation.index | SCIE | * |
dc.relation.index | SCOPUS | * |
dc.relation.index | KCI | * |
dc.relation.startpage | 1185 | * |
dc.relation.lastpage | 1190 | * |
dc.relation.journaltitle | Journal of Korean Medical Science | * |
dc.identifier.doi | 10.3346/jkms.2011.26.9.1185 | * |
dc.identifier.wosid | WOS:000295191500011 | * |
dc.identifier.scopusid | 2-s2.0-80052807566 | * |
dc.author.google | An H.R. | * |
dc.author.google | Park S. | * |
dc.author.google | Yoo T.-H. | * |
dc.author.google | Kang S.-W. | * |
dc.author.google | Ryu J.-H. | * |
dc.author.google | Lee Y.K. | * |
dc.author.google | Yu M. | * |
dc.author.google | Ryu D.-R. | * |
dc.author.google | Kim S.J. | * |
dc.author.google | Kang D.-H. | * |
dc.author.google | Choi K.B. | * |
dc.contributor.scopusid | 강덕희(17233695600) | * |
dc.contributor.scopusid | 최규복(36096388100) | * |
dc.contributor.scopusid | 김승정(8619054500) | * |
dc.contributor.scopusid | 류동열(7103144218;56997547400;56669926200) | * |
dc.date.modifydate | 20240419142141 | * |