Full metadata record
DC Field | Value | Language |
---|---|---|
dc.contributor.author | 이진화 | * |
dc.date.accessioned | 2016-08-28T12:08:06Z | - |
dc.date.available | 2016-08-28T12:08:06Z | - |
dc.date.issued | 2011 | * |
dc.identifier.issn | 1027-3719 | * |
dc.identifier.other | OAK-7858 | * |
dc.identifier.uri | https://dspace.ewha.ac.kr/handle/2015.oak/221860 | - |
dc.description.abstract | BACKGROUND: Chronic obstructive pulmonary disease (COPD) can lead to pulmonary hypertension and cor pulmonale, which are predictors of mortality. OBJECTIVE: To identify predictors of increased pulmonary artery pressure (PAP) in COPD patients without resting hypoxaemia, and to characterise COPD patients with increased PAP. DESIGN: A study of 117 COPD patients from the Korean Obstructive Lung Disease (KOLD) cohort who had measurable tricuspid regurgitant flow under transthoracic Doppler echocardiography and no resting hypoxaemia. RESULTS: The mean patient age was 67 years. Mean forced expiratory volume in 1 second (FEV1) was 47% predicted, mean haemoglobin (Hb) concentration was 145 g/l and mean systolic PAP (sPAP) was 33 mmHg. Multiple linear regression analysis showed that Hb was the only factor independently associated with sPAP (beta = -1.752, P = 0.005). Cluster analysis using FEV 1% predicted, sPAP and Hb concentration as variables indicated three patient clusters: Cluster 1 (n = 36; mean FEV 1 44% predicted, mean sPAP 39 mmHg, mean Hb 132 g/l), Cluster 2 (n = 45; FEV 1 35% predicted, sPAP 31 mmHg, Hb 154 g/l), and Cluster 3 (n = 36; FEV 1 65% predicted, sPAP 29 mmHg, Hb 148 g/l). CONCLUSION: Elevated PAP was linked to low haemoglobin levels in COPD without resting hypoxaemia. © 2011 The Union. | * |
dc.language | English | * |
dc.title | Pulmonary artery pressure in chronic obstructive pulmonary disease without resting hypoxaemia | * |
dc.type | Article | * |
dc.relation.issue | 6 | * |
dc.relation.volume | 15 | * |
dc.relation.index | SCI | * |
dc.relation.index | SCIE | * |
dc.relation.index | SCOPUS | * |
dc.relation.startpage | 830 | * |
dc.relation.lastpage | 837+i | * |
dc.relation.journaltitle | International Journal of Tuberculosis and Lung Disease | * |
dc.identifier.doi | 10.5588/ijtld.10.0598 | * |
dc.identifier.wosid | WOS:000293724800022 | * |
dc.identifier.scopusid | 2-s2.0-79955845689 | * |
dc.author.google | Lee J.H. | * |
dc.author.google | Oh Y.-M. | * |
dc.author.google | Seo J.B. | * |
dc.author.google | Lee Y.K. | * |
dc.author.google | Kim W.J. | * |
dc.author.google | Sheen S.S. | * |
dc.author.google | Kim T.-H. | * |
dc.author.google | Lee J.-H. | * |
dc.author.google | Kim E.-K. | * |
dc.author.google | Lee J.S. | * |
dc.author.google | Huh J.W. | * |
dc.author.google | Lim S.Y. | * |
dc.author.google | Yoon H.I. | * |
dc.author.google | Shin T.R. | * |
dc.author.google | Lee S.-M. | * |
dc.author.google | Lee S.Y. | * |
dc.author.google | Lee S.-D. | * |
dc.contributor.scopusid | 이진화(56646645800;58376333800) | * |
dc.date.modifydate | 20240419140935 | * |