Full metadata record
DC Field | Value | Language |
---|---|---|
dc.contributor.author | 이향운 | * |
dc.date.accessioned | 2016-08-28T12:08:50Z | - |
dc.date.available | 2016-08-28T12:08:50Z | - |
dc.date.issued | 2010 | * |
dc.identifier.issn | 1388-2457 | * |
dc.identifier.other | OAK-6287 | * |
dc.identifier.uri | https://dspace.ewha.ac.kr/handle/2015.oak/220546 | - |
dc.description.abstract | Objective: To study when afterdischarges (ADs) are more likely to occur during cortical stimulation. Methods: We examined 6250 electrical stimulation trials in 13 patients with subdural electrodes, studying whether AD occurrence during a trial was influenced by electrode pair stimulated or AD occurrence during the previous trial. In total 545 electrodes were stimulated, 119 frontal (pre-perirolandic), 289 perirolandic, 36 parietal (post-perirolandic), 95 temporal, and 6 occipital. Results: When the same electrode pair was stimulated as the prior trial, 19% produced ADs compared to 5% of trials when a different electrodes pair was stimulated (p < 0.0001). When trials showed ADs, and the next trial stimulated the same electrode pair, ADs occurred in 46% of cases, compared to 13% of trials following trials without ADs (p < 0.0001). AD probability decreased with increased inter-trial interval length only when the prior trial was at the same electrode pair and had produced an AD (p = 0.001). AD probability increased with stimulation duration, whether the trial followed a trial with (p < 0.001) or without (p < 0.0001) an AD. Conclusions: ADs were more likely to occur when an electrode pair showed ADs and was stimulated again, especially when stimulating after short inter-trial intervals or for longer duration. Significance: When ADs occur, waiting about a minute before resuming stimulation might lessen the likelihood of AD recurrence. © 2009 International Federation of Clinical Neurophysiology. | * |
dc.language | English | * |
dc.title | When is electrical cortical stimulation more likely to produce afterdischarges? | * |
dc.type | Article | * |
dc.relation.issue | 1 | * |
dc.relation.volume | 121 | * |
dc.relation.index | SCI | * |
dc.relation.index | SCIE | * |
dc.relation.index | SCOPUS | * |
dc.relation.startpage | 14 | * |
dc.relation.lastpage | 20 | * |
dc.relation.journaltitle | Clinical Neurophysiology | * |
dc.identifier.doi | 10.1016/j.clinph.2009.10.001 | * |
dc.identifier.wosid | WOS:000274557500005 | * |
dc.identifier.scopusid | 2-s2.0-73449105019 | * |
dc.author.google | Lee H.W. | * |
dc.author.google | Webber W.R.S. | * |
dc.author.google | Crone N. | * |
dc.author.google | Miglioretti D.L. | * |
dc.author.google | Lesser R.P. | * |
dc.contributor.scopusid | 이향운(34667959700) | * |
dc.date.modifydate | 20240123091326 | * |