Full metadata record
DC Field | Value | Language |
---|---|---|
dc.contributor.author | 이원재 | - |
dc.date.accessioned | 2016-08-28T12:08:02Z | - |
dc.date.available | 2016-08-28T12:08:02Z | - |
dc.date.issued | 2008 | - |
dc.identifier.issn | 1931-3128 | - |
dc.identifier.other | OAK-5040 | - |
dc.identifier.uri | https://dspace.ewha.ac.kr/handle/2015.oak/220058 | - |
dc.description.abstract | Although gut epithelia are in constant contact with a large number of microbe-derived immune elicitors such as peptidoglycan, they do not mount an intense immune response. In this issue of Cell Host & Microbe, Lhocine et al. (2008) report that the PIMS gene product induces immune tolerance by reducing the availability of the peptidoglycan receptor in the plasma membrane. These findings provide a novel insight into the molecular mechanisms by which the host gut maintains balanced host-microbe interactions. © 2008 Elsevier Inc. All rights reserved. | - |
dc.language | English | - |
dc.title | How Do Flies Tolerate Microorganisms in the Gut? | - |
dc.type | Short Survey | - |
dc.relation.issue | 2 | - |
dc.relation.volume | 4 | - |
dc.relation.index | SCI | - |
dc.relation.index | SCIE | - |
dc.relation.index | SCOPUS | - |
dc.relation.startpage | 91 | - |
dc.relation.lastpage | 93 | - |
dc.relation.journaltitle | Cell Host and Microbe | - |
dc.identifier.doi | 10.1016/j.chom.2008.07.007 | - |
dc.identifier.wosid | WOS:000258941500003 | - |
dc.identifier.scopusid | 2-s2.0-48649096396 | - |
dc.author.google | Lee W.-J. | - |
dc.contributor.scopusid | 이원재(57171092600) | - |
dc.date.modifydate | 20211210153636 | - |